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Further research using BNC1 / mice demonstrated that BNC1 defects promote intracellular iron accumulation and induce oocyte ferroptosis by activating the Merlin (NF2)/Hippo/YAP/TFRC/ACSL4 pathway (Wang F

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Studies have shown that systemic OGP administration increases bone formation rate and trabecular bone volume in intact animals and accelerates fracture healing in models of skeletal injury

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