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10.1002/jcph.1008 87 CollinF

Hyperoxidized (sulfinic and sulfonic acids) Prx3 was suggested as a ferroptosis marker [156]

R Clinical implications of slow acetylation: Increased risk of isoniazid-induced hepatotoxicity and peripheral neuropathy (tuberculosis treatment) R Increased risk of sulfonamide hypersensitivity reactions R Increased risk of aromatic amine-related bladder cancer in occupational exposure settings (dye workers, rubber industry) R Drug-induced lupus from hydralazine and procainamide occurs almost exclusively in slow acetylators R Altered caffeine metabolism (slow acetylators clear caffeine more slowly via the NAT2 pathway) R NAT1 polymorphisms are less well characterized but the slow allele NAT1*14 has been associated with increased risk of cancers from dietary aromatic amines

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It upregulates growth factor expression (including EGF and VEGF), modulates nitric oxide pathways, and counteracts the damage caused by NSAIDs, alcohol, and chronic stress on the stomach and intestinal walls