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Further supporting this, Lock and colleagues demonstrated that glutathione protects against 2-chloropropionic acid (CPA)-induced cerebellar toxicity by preserving thiol redox equilibrium and potentially interacting with NMDA receptor-mediated pathways
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J Clin Periodontol 24:287296 Heath PR, Shaw PJ (2002) Update on the glutamatergic neurotransmitter system and the role of excitotoxicity in amyotrophic lateral sclerosis
Tells pituitary to release GH
Levocarnitine promotes mitochondrial lipid oxidation in liver cells, reducing the accumulation of fat within hepatocytes
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