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Epigenetic and Cellular Reprogramming: By blocking NNMT-mediated methylation of nicotinamide, preserves NAD salvage pathway activity, increases SIRT-1 signaling, reduces histone deacetylation, and promotes metabolic reprogramming toward oxidative phosphorylation and glycolysis balance, supporting tissue repair and energy homeostasis 1-4

Once in the dermis, GHK-Cu interacts directly with fibroblasts and keratinocytes

This is a significant advantage, as many substances cant get where they need to go

No human clinical trials exist

Myelin protects nerve fibers

Instead, we use forms that are already biologically active and ready for cellular use