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S-1-propenylcysteine improves TNF--induced vascular endothelial barrier dysfunction by suppressing the GEF-H1/RhoA/Rac pathway

The enzyme dipeptidyl peptidase-4 (DPP-4) rapidly degrades circulating GLP-1, which is why pharmaceutical versions are chemically modified to resist this breakdown and maintain therapeutic levels for extended periods

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The possible mechanisms are as follows